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Curcumin, while poorly absorbed in its native form, undergoes significant metabolic transformation in the liver, resulting in metabolites that are excreted in the urine and contribute to its anti-inflammatory effects

In contrast, markers of processing POMC into ACTH and of ACTH secretion, known to be negatively regulated by glucocorticoid receptor ligand binding, were suppressed at all time points, offering explanation for the low ACTH and the high POMC plasma levels